a) that in CFS mitochondrial function is impaired.
b) that in 9/14 people with CFS they could test so far the cause for this impairment is an overproduction of a protein called WASF3 interfering with energy production.
So now we do know a molecular mechanism that causes some types of CFS. This elevates (some types of) CFS from the status of a nebulous clinical syndrome to a metabolic disorder.
Give it another 20 years if well funded and this might eventually turn into a test for these types of CFS or even a treatment. (Which may or may not be more effective as what we habe already).
They found:
a) that in CFS mitochondrial function is impaired. b) that in 9/14 people with CFS they could test so far the cause for this impairment is an overproduction of a protein called WASF3 interfering with energy production.
So now we do know a molecular mechanism that causes some types of CFS. This elevates (some types of) CFS from the status of a nebulous clinical syndrome to a metabolic disorder.
Give it another 20 years if well funded and this might eventually turn into a test for these types of CFS or even a treatment. (Which may or may not be more effective as what we habe already).